Seri Kembangan, Malaysia
#5225 Feb 25, 2013
The Specific Carbohydrate Diet may be helpful for people with Crohns disease. According to Elaine Gottschall in her book, Breaking the Vicious Cycle, the presence of undigested and unabsorbed carbohydrates within the small intestine can encourage microbes from the colon to take up residence in the small intestine and to continue to multiply. This in turn may lead to the formation of lactic and acetic acid, in addition to gas, which injure the small intestine. The principle behind this diet is to maximally nourish the individual and minimally nourish the intestinal microbes. Therefore, by specifically eating only mono-saccharides which require minimal digestion and which are absorbed well it theoretically leaves nothing to be used by the microbes to further their growth in the intestines. According to her theory the microbial population will decrease due to lack of food and thus their harmful by-products will also decrease freeing the intestinal surface of injurious substances. No longer needing protection, the mucus-producing cells of the intestine stop producing excessive mucus and carbohydrate digestion is improved.
On The Specific Carbohydrate Diet you are not allowed to eat sugars, alcohol, grains, dairy products, legumes, potatoes, yams or parsnips. Foods which are allowed on this diet include beef, lamb, poultry, fish, eggs, natural cheeses, homemade yogurt, fruit, nuts, pure fruit juices, weak coffee, weak tea and peppermint and spearmint teas. Also allowed are corn, soy, safflower, sunflower and olive oils.
Treatment: In addition to the recommendations above Barbara and I get very good results treating Crohns disease and ulcerative colitis using acupuncture, gentle hara visceral work, dietary and lifestyle changes, and specific herbs and supplements aimed at restoring the natural balance in the colon. We recommend that our patients suffering from colon problems eat lightly in the evening when the colon has more difficulty in the digestion and transit of foodstuffs. Avoid fat, meat and sugar as these are most likely to cause problems. During treatment it is better that you ingest food with long fibers such as leeks, spinach, celery and Swiss chard in order to improve the general function of the colon. Like the small intestine, the large intestine is dependent on the liver and pancreas, therefore it is a good idea to stimulate these organs with olive oil and lemon during the course of treatment. We may also recommend certain herbs you may take for this.
Seri Kembangan, Malaysia
#5226 Feb 25, 2013
Chronic diarrhea is often a sign of a parasitic infection. Other symptoms include pain, constipation, bloating, gas, unexplained weight loss, fatigue, unexplained fever, coughing, itching, rashes, bloody stools, abdominal cramping, joint and muscle aches, irritable bowel syndrome, anemia, allergies, granulomas, nervousness, teeth grinding, chronic fatigue, poor immune response, and sleep disturbances. These symptoms can come and go due to the life cycles of the specific parasite involved.
Useful herbs for parasitic infestations:
♦ Garlic has antiviral, antibacterial and antifungal properties. Allicin, the active ingredient in garlic, has been shown to be effective against E. histolytica and Giardia lamblia.
♦ Goldenseal has been shown to be effective against amebas and Giardia parasites.
♦ Grapefruit Seed Extract - is a general antimicrobial agent with specific antibacterial, antiviral, antiparasitic and anti yeast properties. It is effective against E. Coli, Salmonella typhi, Staph aureus, E. histolytica, Giardia lamblia, herpes simplex type 1 and influenza A virus.
R. IRRITABLE BOWEL SYNDROME
What is Irritable Bowel Syndrome?
Irritable Bowel Syndrome (IBS) is a disease of the entire GI tract in which persons may react to stress by developing symptoms such as cramping and abdominal pain associated with alternating patterns of diarrhea and constipation. The episodes of diarrhea are often accompanied by crampy abdominal pains or gas which are relieved by a bowel movement. Excessive amounts of mucus may appear in the stools. Other symptoms include flatulence, spasms, bloating, gas, abnormal bowel movements, nausea and loss of appetite. After the diarrhea one may be plagued by long bouts of constipation. One's stool may also contain mucus because in IBS the colon secretes too much mucus. However, blood mixed with the stools is not IBS but something else. Fever, weight loss or severe pain are not signs of IBS,
Irritable bowel syndrome affects 10 - 20% of all American adults and is the most common gastrointestinal complaint. After the common cold IBS is the chief cause of absenteeism. About 60% of IBS sufferers report their symptoms first coincided with stressful life events. Many people find that their symptoms persist well beyond the stressful life event and the episodes invade their normal routine
There is no single cause of IBS. Some scientists believe a virus or bacterium may play a role. Lifestyle factors such as stress, nervousness and diet are probably common causes. The overuse of antibiotics, antacids or laxatives which disturb the bacterial microflora of the bowel may also be a factor.
Over 100 different disorders may be linked to the systemic effects of IBS. One disorder that is linked in about 25% of adults with IBS is arthritis which affects the ankles, knees and wrists. IBS has been linked to a higher than normal incidence of colon cancer and diverticulitis. If IBS causes chronic diarrhea, electrolyte and trace mineral deficiencies are likely,
Diagnosis of IBS requires ruling out disorders that can cause similar symptoms such as Crohn's disease, diverticulitis and ulcerative colitis.
Seri Kembangan, Malaysia
#5227 Feb 25, 2013
Whole grains, vegetables, legumes and sprouts. In general, consuming a low fat diet rich in complex carbohydrates and dietary fiber is often curative. Eat small meals and chew food well.
It is beneficial to cook with organic butter or ghee (clarified butter) because butter and ghee supply butyrate to the colon. Butyrate acts as a fuel molecule for the colon. All the energy for the colon cells comes from butyrate.
A good diet for someone whose GI tract is stressed out is rice and split moong dal beans. Boil the moong dal. Sauté in ghee, organic butter or olive oil, some chopped onions, chopped ginger and coriander, a pinch of cumin seeds, a pinch of turmeric, a pinch of black pepper, and a pinch of cayenne. Once the onions are soft then transfer the mixture to the dal. Then cook them together for 10 mins. Add salt to taste. Cook the rice separately. Use Basmati white rice. Brown rice has too much fiber on it. You can eat any vegetable as long as you cook it. Asparagus, boiled potatoes, carrots and green peas are good, but corn, for example, is hard to digest.
Foods to avoid:
The most common foods that trigger IBS are wheat, corn and dairy products, coffee, tea, citrus fruits and chocolate. It is also advisable to avoid animal fat, butter which is not organic, all carbonated beverages, candy, fried foods, all junk foods, the additives mannitol and sorbitol, margarine, nuts, pastries, all processed foods, seeds, spicy foods, sugar, and sugar-free chewing gum. These foods encourage the secretion of mucus by the membranes and prevent the uptake of nutrients. Check to see if you have food allergies. They are important factors in this disorder. Food sensitivities are found in one half to two-thirds of people with IBS. An elimination diet is a good way to check to see if you have food allergies or food sensitivities.
It is better to avoid excess consumption of meat, chicken or fish as the body converts any amino acid (which come from the proteins in the meat, etc.) in the liver into urea. Then the urea goes into the colon and the bacteria in the colon convert it into ammonia. Ammonia is used up in many ways but if an excess is produced then it destroys the mucus of the lining of the large intestine.
Alcohol and tobacco should be avoided as they irritate the linings of the stomach and colon
Monitor your intake of the cabbage family of foods (broccoli, cauliflower and Brussel sprouts) as they can be difficult to digest.
An increase in physical exercise appears helpful for IBS patients suffering from stress. Daily leisurely walks markedly reduce symptoms for many people.
Chew your food well. Do not overeat or eat in a hurry
Wait at least one or two hours after eating before going to bed. And eat lightly in the evening.
Seri Kembangan, Malaysia
#5228 Feb 25, 2013
Lactose intolerance is the impaired ability to digest lactose a sugar naturally found in milk products. Individuals with this condition have a deficiency in the enzyme lactase which digests lactose. Symptoms occur when food is consumed containing lactose. These symptoms range from cramps, gas, and diarrhea to acne, bloating, headaches and nausea.
Although symptoms of lactose intolerance are triggered by the lactose in some dairy products, few lactose-intolerant individuals need to avoid all dairy. Dairy products have varying levels of lactose, which affects how much lactase is required for proper digestion.
How to test for Lactose Intolerance:
There are two ways to test for it -- one is a laboratory test and the other is a self-test. To do a self-test you restrict intake of all dairy products for at least ten days. Restricted foods include: milk, yogurt, ice cream, cheese, creamed soups, frozen yogurt, powdered milk, whipped cream, as well as bakery items, cookies, hot dogs, lunchmeats, milk chocolate, nondairy creamers, pancakes, protein powder drinks and ranch dressings. If lactose intolerance is causing your problems you will probably notice your symptoms will disappear significantly. Reintroduction of dairy products will trigger a return of symptoms. However, the results may be inconclusive because you may be sensitive to other foods in which case your symptoms wont change. In this case you could either do an elimination diet to find out what other foods you are sensitive to or you could do a laboratory test.
Treatment: Supplementation with the enzyme lactase can be helpful to prevent symptoms of lactose intolerance when consuming lactose-containing dairy products. Because the degree of lactose intolerance varies from individual to individual a greater or lesser amount of lactase may be needed to eliminate the symptoms.
T. Leaky Gut Syndrome
Leaky Gut Syndrome also known as Increased Intestinal Permeability occurs when foods are not digested properly and ferment and rot in our intestines producing toxic by-products that irritate and inflame the intestinal lining causing tiny holes. When the intestinal lining is damaged even more, substances larger than particle size disease-causing bacteria, potentially toxic molecules, and undigested food particles are allowed to pass directly through the weakened cell membranes. They go directly into the blood stream activating antibodies and cytokines. The cytokines alert our white blood cells to battle the particles. Oxidants are produced in the battle, causing irritation and inflammation far from the digestive system.
Symptoms associated with Leaky Gut Syndrome include abdominal pain, indigestion, diarrhea, constipation, bloating, gas, chronic joint and muscle pain, confused and fuzzy thinking, poor memory, mood swings, nervousness, anxiety, poor immunity, recurrent vaginal and bladder infections, shortness of breath, fatigue and malaise.
Causes of Leaky Gut Syndrome include chronic stress, dysbiosis, environmental contaminants, GI tract disease, immune system overload, excessive alcohol consumption, low fiber diet, highly processed foods, existence of pathogenic bacteria, parasites and yeasts and prolonged use of NSAIDs, birth control pills, steroid drugs, chemotherapy and radiation therapy
Leaky gut syndrome is associated with allergies, celiac disease, Crohns disease and malabsorption syndromes. It is also linked to autoimmune diseases like ankylosing spondylitis, asthma, bronchitis, eczema, psoriasis, food and environmental sensitivities, rheumatoid arthritis and Sjogrens syndrome.
Seri Kembangan, Malaysia
#5229 Feb 25, 2013
♦ Chew your food thoroughly.
♦ If you have food allergies or sensitivities avoid those foods which cause them.
♦ If you have dysbiosis or Candidiasis get appropriate treatment.
♦ Use of antioxidants such as vitamin E, selenium, N-acetyl cysteine, superoxide dismutase, zinc, manganese, copper, Coenzyme Q10, lipoic acid and vitamin C can help quench the flare up of free radicals often produced when there has been damage to the intestinal tract.
♦ Glutamine may be especially helpful in repairing the mucosal lining directly as it is the preferred food of the cells of the small intestine. Dosage: 1 gm to 30 gm daily.
♦ Other useful nutrients and herbs include: gamma-oryzanol, vitamin A, vitamin B5, DGL (deglycyrrhized licorice), folic acid and schizandra.
♦ In addition to the above recommendations Barbara and I get very good results using acupuncture and gentle hara visceral work to restore balance and normal function to the GI tract.
U. pancreatic insufficiency
Pancreatic insufficiency is common in people with candidiasis or parasite infections. Pancreatic enzymes help digest protein, fats, carbohydrates and the fat soluble vitamins E, A, K and D.
Symptoms of low pancreatic enzymes and malabsorption
♦ Gas, bloating and indigestion ½ hour to several hours after eating.
♦ Chronic excessive intestinal gas.
♦ Chronic constipation, sometimes mild diarrhea or alternating constipation and diarrhea.
♦ Frequent undigested food in the stools
♦ Floating or bubbling stools (because gas and grease float).
♦ Fat soluble vitamin deficiency signs.
♦ Taking supplements or eating properly and not getting corresponding improvement in health.
♦ Pancreatic enzyme supplements work well as a digestive aid. CAUTION: Do not take pancreatic enzymes if you have ulcers or any kind of inflammatory bowel condition.
♦ Vegetable enzymes work in a much wider range of pH than pancreatic enzymes, enhancing digestion in the stomach as well as in the intestines. And they are less likely to cause food allergy reactions than pancreatic enzymes. Dosage: one to two capsules at the beginning of meals.
Seri Kembangan, Malaysia
#5230 Feb 25, 2013
peptic Ulcers: gastric and duodenal ulcers
In the US about 5 to 10% of the population develops peptic ulcers. An ulcer is a craterlike lesion in a membrane. Ulcers that develop in areas of the GI tract exposed to acidic gastric juice are called peptic ulcers. Most occur on the lesser curvature of the stomach where they are called gastric ulcers, or in the pyloric sphincter of first part of the duodenum, where they are called duodenal ulcers. Most peptic ulcers are duodenal.
The most common symptom is epigastric pain that is burning or gnawing in nature. Typically, the pain occurs 1 ½ to 3 hours after eating and is relieved within a few minutes by ingestion of food.
According to Elizabeth Lipski in her book, Digestive Wellness, about 10% of stomach and duodenal ulcers are caused by the use of NSAIDs such as aspirin, Tylenol, Motrin and prescription pain relievers. It has been estimated that 107,000 people are hospitalized each year due to NSAID complications, and at least 16,5000 NSAID-related deaths occur each year among arthritis patients alone. It has now been found that 80% of people with duodenal and stomach ulcers and gastritis have a bacteria called Heliobacter pylori (H. pylori). H. pylori is found in about 30% of people, but only 10% will experience ulcers. And 10% of people with ulcers and gastritis do not have H. pylori, nor have they used NSAIDs. What is known, however, is that when H. pylori is eradicated, ulcers heal and dont recur.
Seri Kembangan, Malaysia
#5231 Feb 25, 2013
~~ Quick eradication of H. pylori bacteria is important for healing of ulcers. There are some herbal remedies you can try first if you want to avoid antibiotics.
~~ DGL (deglycyrrhizinated licorice) is a wonderful healing aid for ulcers. It increases the production of protective mucus in the stomach. Numerous studies over the years have found DGL to be an effective anti-ulcer compound. DGL has been shown to be as effective as Tagamet in both short term treatment and maintenance therapy of gastric ulcer. In another study it was as effective as Zantac in the treatment of gastric ulcer. There are no known side effects to using DGL. Dosage: Chew one or two wafers (300 mg each) before meals.
~~ Unripe banana, slippery elm, marshmallow root and raw cabbage juice have anti-ulcer effects.
~~ Avoid tobacco, coffee, alcohol and the use of NSAIDs if you have any type of ulcer.
~~ Avoid refined sugars, fried foods, white-flour products, milled maize, chocolate, soft drinks, desserts, heated or poor quality vegetable oils, red meats, hot spices, vinegar and citrus fruit.
~~ Eat soothing mucilaginous foods and preparations, such as soups, rice congee, avocado, spinach, cucumber, potato, lettuce and soothing herbal teas.
~~ Other remedies used include citrus seed extract, goldenseal, activated charcoal and aloe vera.
~~ One successful dietary approach has been insuring a good intake of vitamin A followed by a high-fiber diet and seven or more servings of fruits and vegetables per day.
Seri Kembangan, Malaysia
#5234 Feb 25, 2013
Glutamine Supplements Show Promise In Treating Stomach Ulcers
May 22, 2009 Nearly 20 years ago, it was discovered that bacteria known as Helicobacter pylori were responsible for stomach ulcers. Since then, antibiotics have become the primary therapy used to combat the H. pylori infection, which affects approximately six percent of the world population and is also a primary cause of stomach cancer. But today the bacteria is growing increasingly resistant to antibiotics.
Now a study led by scientists at Beth Israel Deaconess Medical Center (BIDMC) and the Massachusetts Institute of Technology demonstrates that the amino acid glutamine, found in many foods as well as in dietary supplements, may prove beneficial in offsetting gastric damage caused by H. pylori infection. Reported in the May 2009 issue of the Journal of Nutrition., the findings offer the possibility of an alternative to antibiotics for the treatment of stomach ulcers.
"Our findings suggest that extra glutamine in the diet could protect against gastric damage caused by H. pylori," says senior author Susan Hagen, PhD, Associate Director of Research in the Department of Surgery at BIDMC and Associate Professor of Surgery at Harvard Medical School. "Gastric damage develops when the bacteria weakens the stomach's protective mucous coating, damages cells and elicits a robust immune response that is ineffective at ridding the infection." Eventually, she notes, years of infection result in a combination of persistent gastritis, cell damage and an environment conducive to cancer development.
Glutamine is a nonessential amino acid naturally found in certain foods, including beef, chicken, fish, eggs, dairy products and some fruits and vegetables. L-glutamine the biologically active isomer of glutamine is widely used as a dietary supplement by body builders to increase muscle mass.
Hagen and her coauthors had previously shown that glutamine protects against cell death from H. pylori-produced ammonia. "Our work demonstrated that the damaging effects of ammonia on gastric cells could be reversed completely by the administration of L-glutamine," explains Hagen.
"The amino acid stimulated ammonia detoxification in the stomach as it does in the liver so that the effective concentration of ammonia was reduced, thereby blocking cell damage."
She and her coauthors, therefore, hypothesized that a similar mechanism might be at work in the intact stomach infected with H. pylori. To test this hypothesis, the investigators divided 105 mice into two groups, which were fed either a standardized diet (containing 1.9 percent glutamine) or the same diet with supplemental L-glutamine (containing 6.9 percent glutamine) replacing carbohydrates for five percent of the total calories.
Seri Kembangan, Malaysia
#5236 Feb 25, 2013
Glutamine Protects GI mucosal from H pylori and ammonia toxicity
In another experiment glutamine or marzulene (a gastroprotective drug containing 1% sodium azulene and 99% glutamine) protects the gastric mucosa against H. pylori in vivo and investigated the mechanism underlying glutamine-induced mucosal protection against ammonia in gastric epithelial cells in vitro. Mongolian gerbils were fed for 3 months with a diet containing glutamine (2%-20%) or marzulene (20%) starting from 2 weeks or 2 years after H. pylori infection. Then, gastric mucosal changes were evaluated both macro- and microscopically. Cultured gastric epithelial cells were incubated in the presence of ammonia, with or without glutamine; and cell viability, ammonia accumulation, and chemokine production were determined. Gerbils exhibited edema, congestion, and erosion after 3-month infection; and after 2-year infection, they showed cancer-like changes in the gastric mucosa. Glutamine and marzulene significantly suppressed these pathological changes caused in the gastric mucosa by H. pylori infection.
Ammonia was accumulated in the cells, resulting in an increase in chemokine production and a decrease in cell viability. These pathological responses were prevented by glutamine. In addition, glutamine decreased chemokine production and cell death through inhibition of cellular accumulation of ammonia, resulting in the prevention of H. pylori-induced gastric diseases in vivo. These results suggest that glutamine/marzulene would be useful for prophylactic treatment of H. pylori-induced gastric diseases in patients.
Seri Kembangan, Malaysia
#5238 Feb 25, 2013
Glutamine is the most popular anti-ulcer drug in Asia today. The digestive tract uses glutamine as a fuel source and for healing. It is effective for healing stomach ulcers, IBS and ulcerative bowel diseases. Dosage: Begin with 8 grams daily for a trial period of 4 weeks.
Goldenseal is soothing to mucus membranes, enhances immune function and has antibiotic and antifungal properties.
Multivitamin and mineral complex - supplies those nutrients lost or not absorbed.
Oat bran fiber and psyllium seeds - have healing and cleansing effects
Proteolytic enzymes with pancreatin - aid in protein digestion and in reducing inflammation. Use a formula that is low in HCl and high in pancreatin.
Slippery Elm - is a remarkably effective GI soother. It has demulcent properties and has been a folk remedy for both heartburn and ulcers.
Vitamin A is protective and promotes healing of gastric ulcers. Dosage: 10,000-25,000 IU daily. CAUTION: Pregnant women should not exceed 10,000 IU of vitamin A
Vitamin B complex - is needed for proper muscle tone in the gastrointestinal tract.
Zinc increases the rate of healing and can prevent damage to the stomach lining. Dosage: 50-100 mg daily.
Broad spectrum anti-microbial herbs:
Clove - possesses strong and proven antimicrobial properties against fungus, bacteria and parasites.
Echinacea - contains an antibiotic compound with broad spectrum activity.
Forsythia - is an outstanding antiviral used for colds and flus.
Garlic - has antibacterial, antifungal, antiviral and antiparasitic properties.
Grapefruit Seed Extract - is a general antimicrobial agent with specific antibacterial, antiviral, antiparasitic and anti yeast properties. It is effective against E. Coli, Salmonella typhi, Staph aureus, E. histolytica, Giardia lamblia, herpes simplex type 1 and influenza A virus.
Isatis - is one of the best known Chinese antiviral herbs. It is an excellent remedy for any virus. It is mild and can be used for children. It is also a good antibacterial agent.
Pau d'arco - excels in controlling Candida and yeast overgrowth. It also exhibits antiviral and antibacterial properties.
Skullcap (Scutellaria baicalensis)- has broad spectrum antimicrobial activity and it suppresses viruses.
Seri Kembangan, Malaysia
#5239 Feb 25, 2013
NUTRITIONAL & BOTANICAL SUPPORT FOR MIGRAINES
· Feverfew*(Tanacetum parthenium) has anti-inflammatory properties
· Boswellia*- has anti-inflammatory properties. It is a LOX-5 inhibitor as well as a COX-2, 5, 12 and 15 inhibitor.
· Parthenolide*- inhibits certain pro-inflammatory pathways such as NF-kappaB
· Andrographis*- is anti-inflammatory. It downregulates the pro-inflammatory cytokines NF-kappaB and IL-1 beta.
· Omega 3 Fatty Acids have anti-inflammatory properties
· Willow Bark has anti-inflammatory properties
· Resveratrol has anti-inflammatory properties
· Turmeric (Curcumin) has anti-inflammatory properties
· CoQ10 increases blood flow to the brain and improves circulation.
· Magnesium and Calcium help to regulate muscular tone; magnesium supplementation may be beneficial for migraines.
· Quercetin has anti-inflammatory properties
· Cats Claw has anti-inflammatory properties
· Atractylodes has anti-inflammatory properties
· Grape Seed proanthocyanidins have anti-inflammatory properties
· Green Tea Extract has anti-inflammatory properties
· Honokiol* has anti-inflammatory properties
· Magnolol* has anti-inflammatory properties
· Milk Thistle (Silymarin) has anti-inflammatory properties
· Panax ginseng & Panax notoginseng have anti-inflammatory properties
· Holy Basil & Rosemary have anti-inflammatory properties
· Chamomile, peppermint & lemon balm tea are very relaxing
· Reishi mushrooms have anti-inflammatory properties
· Chinese skullcap* has anti-inflammatory properties
· Royal Jelly extract has anti-inflammatory properties
Seri Kembangan, Malaysia
#5240 Feb 25, 2013
Butterbur A double-blind, placebo-controlled study showed that the total number of days of migraine pain were significantly reduced by butterbur compared to the placebo group.(Grossmann, 2000) Another study showed a 56% reduction in migraines using butterbur.(Eaton 1998)
Feverfew In a systematic review by Ernst & Pittler (2000) they concluded that feverfew is likely to be effective in the prevention of migraine and that there are no major safety problems.
Calcium & Magnesium - help to regulate muscular tone and to transmit nerve impulses throughout the body and to the brain. Use chelate forms.
Magnesium may be beneficial for FMS migraines. Magnesium supplementation may be beneficial for migraines.
Cayenne, chamomile. ginger, peppermint, rosemary, valerian and willow bark - are effective herbs in the treatment of migraines.
Coenzyme Q10 - Increases blood flow to the brain and improves circulation. CoQ10 has shown promise as a migraine preventive agent.(Rozen, 2000)
Essential fatty acids - needed for brain cells and for fat metabolism. Take Vit. E when taking EFAs.
Ginkgo biloba extract - enhances cerebral circulation.
Lecithin - helps to reduce the frequency and severity of migraines.
Natural progesterone cream - may benefit women who suffer from migraines.
Vitamin B2 -(Riboflavin)- at a dose of 400 mg per day can significantly reduce the frequency and duration of migraines according to preliminary research. This dose, which appears to be safe, is much higher than what you'll find in either foods or a vitamin supplement so you should discuss B2 therapy with your physician.(Dr. Andrew Weil's Self Healing Newsletter. April 2000).
For general information on herbs and supplements which can relieve pain please read our article entitled Pain Management.
NUTRITIONAL & BOTANICAL SUPPORT FOR HEADACHES
Bromelain - helps to regulate the inflammatory response.
5-HTP is useful for tension headache prophylaxis.(Songo et al, 1984)
Calcium and magnesium - help to alleviate muscular tension. Use chelate forms.
Coenzyme Ql0 - increases blood flow to the brain and improves circulation.
Ginkgo biloba extract - enhances cerebral circulation
Feverfew, ginger, brigham, burdock root. fenugreek, lavender, marshmallow, mint, rosemary, skullcap and thyme - are herbs which may relieve headache pain.
Primrose oil - supplies essential fatty acids which promote healthy circulation, help regulate the inflammatory response and relieve pain.
Seri Kembangan, Malaysia
#5241 Feb 25, 2013
LIFESTYLE RECOMMENDATIONS FOR MIGRAINE & HEADACHE SUFFERERS
Get regular moderate exercise. Do not smoke and avoid secondhand smoke.
Eat small meals, and eat small, nutritious snacks between meals if needed, to help stabilize wide swings in blood sugar that may precipitate a migraine. Especially avoid missing meals. Elimination of allergenic foods from the diet may help.
The frequent use of over-the-counter painkillers may actually increase the likelihood of migraines.
USEFUL FOODS FOR HEADACHES & MIGRAINES
Almonds, almond milk, watercress, parsley, fennel, garlic, ginger, cherries and fresh pineapple.
FOODS TO AVOID FOR MIGRAINES
Omit foods which contain the amino acid tyramine including alcohol, aged meats, fermented sausages, aged cheeses, sour cream, avocados, bananas, beer, cabbage, canned fish, dairy products, eggplant, chicken, chocolate, potatoes, cold cuts, herring, onions, peanut butter, pork, smoked fish, vinegar, raspberries, red plums, citrus fruits, tomatoes, wine and yeast.
Avoid aspirin, MSG, nitrites (the preservative used in foods) and spicy foods. Avoid salt and acid-forming foods such as meat, cereal, bread and grains. Avoid fried, fatty and greasy foods, and any food that is aged, cured, pickled, soured, yeasty or fermented. Also avoid red wine, figs, dates, raisins, hard cheeses, yogurt, sour cream, herring and sauerkraut.
Foods which contain high amounts of copper (such as chocolate, nuts, wheat germ and shellfish) or which increase the intestinal absorption of copper (such as citrus) are potential migraine headache triggering agents.
Avoid foods which contain sulfites such as white wine and foods that use sulfites as preservatives such as beer, cider, apple juice, mashed potatoes and dried apricots.
Avoid milk chocolate as it is more migrainogenic than dark chocolate.
Estrogen level has been correlated with triggering of migraines. The headache coincides with the drop in estrogen just before the onset of menstruation. Estrogen causes swelling in the brain. The enlarged blood vessels in the brain cause most migraine headaches. Estrogen can also deplete magnesium levels, which makes arteries more susceptible to spasm, a common cause of other types of headaches. According to Dr. John Lee if you have premenstrual headaches, progesterone cream and magnesium will usually cure them within three menstrual cycles.
To find out about some of the side effects of migraine drugs please read our article entitled Pain Management.
FOODS TO AVOID FOR HEADACHES
Tyramine causes the blood pressure to rise, resulting in a dull headache Therefore, try eliminating foods containing tyramine and the amino acid phenylalanine Then reintroduce one food at a time and see which one produces headaches. Phenylalanine is found in aspartame (Equal, NutraSweet), MSG, and nitrites. Foods that contain tyramine are listed above under "Foods to Avoid for Migraines".
Seri Kembangan, Malaysia
#5242 Feb 25, 2013
The pain behind migraines is thought to be due in part to arterial vasodilation, which is often preceded by vasoconstriction. Inflammation of arterial walls, with vasodilation and edema from vasoactive peptides and other pain-inducing substances, also contribute to the intensity of the pain.
The trigeminal nerve carries pain messages from the inflamed arterial wall to the brainstem, triggering migraine pain. Headaches may be related to mechanical irritation of the sensitive nerve endings of the meninges.
About 50 percent of sufferers of migraines have a two to four hour premonitional period before the symptoms appear, during which time they experience vague effects, such as a mild increase in sensitivity to light, loss of appetite, a feeling of being ungrounded, or mild dyslexia.(Craniosacral work is most effective at this stage of the migraine.)
Twenty percent of sufferers see an aura or light display of some kind. The actual headache is accompanied by a throbbing pain, nausea, and light and noise intolerance. A migraine aura reflects some sort of cerebral dysfunction. It begins within 5 - 20 minutes of the migraine and fades away in less than an hour. There are various types of auras. Migraines without auras are preceded by a prodrome including such symptoms as fatigue, sleeplessness, slight behavioral problems and decreased appetite. The migraine appears quickly, and affects the suborbital, frontal and less often the occipital areas. It is one-sided, pulsatile, and often causes photophobia, nausea, vomiting or a state of prostration. The migraine may last from a few hours to three days.
Seri Kembangan, Malaysia
#5243 Feb 25, 2013
In a study done by Sensenig et al (2004) it was concluded that migraines may be, at least in part, caused by an underlying deterioration of normal body functioning. They found that by improving assimilation and elimination mechanisms, the manifestation of migraine diminished or ceased for the majority of the participants.
DESCRIPTION OF OTHER TYPES OF HEADACHES
Non-migraine headaches often begin with nuchal and occipital pain, which migrates slowly toward the anterior cerebral regions, unilaterally or bilaterally, and ends up in the sinuses, frontal, periorbital or more rarely zygomatic region.
Hypertension causes diffuse, predominantly occipital headaches in early evening or early morning. Confusingly, hypotension can cause the same symptoms.
The understanding of headaches has been deepened by the work of brain and orthopedic surgeons, who have documented the sensitivity of soft tissues in the spine, the dural membranes and the brain tissue itself.
CRANIOSACRAL TREATMENT OF MIGRAINES
Specific work with the sphenoid, maxillae, and mandible represents the most powerful way to both prevent and interrupt migraines. Maxillary position and motion, or its absence, seems to have a leading role in migraine causation, making craniosacral vault holds based on Dr. Sutherland's work perhaps the best craniosacral treatments for migraine.
CAUSES OF MIGRAINES ACCORDING TO CHINESE MEDICINE
Emotions - such as frustration, irritability, anger and depression.
Missing meals - weakens the Spleen Qi and allows the Liver Qi to rise upwards.
Alcohol - is Hot and Damp. It enters the Liver making the Liver Hot and enters the Spleen making it Damp, i.e., weak. In addition, alcohol is outwardly and upwardly dispersing and can directly precipitate the venting of Liver Qi.
Chocolate- inflames the Ming Men Fire. Its oils and fats dampen the Spleen and cause Excessive Heat in the Stomach and Liver.
Cheese and milk products - tend to aggravate Dampness and weaken the Spleen at the same time plugging the Qi pathways with Phlegm. This plugging of the pathways increases internal pressure and any tendency to Liver Qi stagnation.
Coffee - disperses Stagnant Qi and wastes the Blood and Yin. So it should not be used by persons who are Stomach Yin Deficient, Blood Deficient or Heart and/or Kidney Yin Deficient. It liberates stored Jing or Essence in the Kidneys and therefore is deleterious to health over-all. It should be used with care by women and the elderly.
Extreme exhaustion - can cause consumption of Blood or a relaxation of the holding Qi which allows pent up Qi to escape.
Smoking - Nicotine is also dispersing and discharging and can precipitate the venting of pent-up Liver Qi. Nicotine constricts blood vessels while carbon monoxide tends to expand them.
CAUSES OF HEADACHES
Common causes of headaches are stress, tension, anxiety, allergies, constipation, coffee consumption, eyestrain, hunger, sinus pressure, muscle tension, hormonal imbalances, TMJ syndrome, trauma to the head, nutritional deficiencies, the use of alcohol, drugs or tobacco, fever and exposure to pollution, perfume or after-shaves.
Headache sufferers may be allergic to certain foods such as: wheat, chocolate, MSG, sulfites, sugar, hot dogs, luncheon meats, dairy products, nuts, citric acid, cheese, sour cream, yogurt, alcohol and vinegar.
Other possible causes of headaches are: anemia, bowel problems, brain disorders, tooth-grinding, high blood pressure, low blood sugar and sinusitis.
Vitamin B6 deficiency, which can cause headaches and migraines, may result from the use of oral contraceptives.
Seri Kembangan, Malaysia
#5244 Feb 25, 2013
Research into the underlying causes of inflammation and its effects on human health has exploded in recent years and much has been discovered and clarified which is giving the medical community a deeper understanding of how inflammation works and how it is connected to many of the deadliest disease of the 21st century. Researchers are discovering that inflammation, the body's basic response to infection and injury, may be at the root of heart disease, diabetes, cancer, asthma, inflammatory bowel disease and Alzheimer's disease.
WHAT IS INFLAMMATION?
o Inflammation is a natural reaction to injury or infection. The affected tissues swell, redden, become warm and tender, and may be painful. Any organ or tissue of the body, internal or external, can become inflamed. During the first stage in inflammation, vasodilation occurs and vascular permeability increases. Both are induced, in large part, by the release of histamine from ruptured or activated mast cells, circulating basophils and platelets. Mast cells also release serotonin, another compound that induces vasodilation.
o Increased vascular permeability leads to local edema, during which phagocytes and humoral factors are released into the intravascular space. Humoral factors include proteins of the complement system (which stimulate mast cells to release histamine and heparin), eicosanoids which are products of arachidonic acid metabolism, kinins (a group of highly bioactive peptides found in a variety of body tissues), cytokines (protein factors produced by cells that regulate local cellular grown and function), and platelet activating factor (which is secreted by activated immune cells).(Boik 1996)
o Eicosanoids act as mediators in heart attack, asthma, arthritis, ulcerative colitis, asthma, dysmenorrhea and recurrent headaches. These inflammatory eicosanoids can be divided into three groups:
v Prostaglandins (PG) primarily have the effects of influencing platelet aggregation and constricting or relaxing blood vessels. Some prostaglandins have favorable effects, such as insuring the integrity of the gastric lining, regulating kidney function and initiating some stages of childbirth, but many produce negative effects, such as intensifying pain, causing cramping and restricting blood circulation. PGE2 is one of the main prostaglandins involved in inflammation.
v Thromboxanes (TX) are produced by platelets; they enhance platelet aggregation and also cause constriction of blood vessels and bronchioles. They are mostly known for their negative impact, particularly on cardiovascular health (TXB) and also as a contributor to asthma (TXA), but they may play a role in the immediate response to injury, to limit blood loss.
v Leukotrienes (LT) are produced by immune system cells and stimulate production of immune system signaling molecules such as interleukins (IL) and interferon, and substances involved in anaphylactic reactions. They are mostly known for their contribution to allergies and autoimmune diseases. Leukotrienes induce bronchospasm; but prostaglandin inhibitors, such as aspirin, can induce asthma attacks, perhaps by leaving more arachidonic acid available for the LOX enzymes. Recently attention has focused on LOX (lipoxygenase) inhibitors that mainly block leukotriene synthesis. 5-LOX appears to be the key enzyme when it comes to blocking leukotriene synthesis.
v While inflammatory responses to acute infection and injuries mediated by these eicosanoids have certain beneficial effects, persistent inflammation has a degrading effect that can lead to debility and death.(Dharmananda 2003)
Seri Kembangan, Malaysia
#5245 Feb 25, 2013
ROLE OF INFLAMMATION IN DISEASE
o Alzheimers Disease
v Giri et al noted that in Alzheimers disease one finds increased deposition of amyloid peptide A beta and also an increased presence of monocytes and macrophages in the vessel wall and activated microglial cells in the brain. Alzheimers disease patients show increased levels of proinflammatory cytokines by activated microglia.(Giri et al 2003)
v According to an article in Alternative Medicine magazine by Guthrie although the mechanism causing Alzheimer's disease is not fully understood, neurologists believe the brain's immune cells rally to attack a form of plaque that signals Alzheimer's disease. The ensuing skirmish creates inflammation that may spur progression of the disease.
v Inflammatory cells, such as activated eosinophils and neutrophils are associated with increased levels of IL-5, IL-8 and of proinflammatory mediators. Viruses, but also endotoxin or allergen exposure, are able to recruit neutrophils, via an IL-8 production by activated macrophages or epithelial cells. Together, these inflammatory mediators are responsible for the diffuse bronchial inflammation. Activated T cells may also be related to the pathogenesis of severe asthma. The persistence of inflammatory cells in bronchi, particularly neutrophils, which respond poorly to corticosteroids, could be in part responsible for the epithelial damage, the extensive mucus plugging, and the abnormalities of epithelial and endothelial permeability which are associated with severe acute asthma.(Tillie-Leblond et al 2005)
v Bronchial provocation with allergen induces a prompt early phase
immunoglobulin E (IgE) mediated decrease in bronchial airflow followed in many patients by a late-phase IgE mediated reaction with a decrease in bronchial airflow for 4-8 hours. Initially, there is recruitment of leukocytes from the bloodstream to the airway by activated DC4 T-lymphocytes. The activated T-lymphocytes also direct the release of inflammatory mediators from eosinophils, mast cells, and lymphocytes. In addition, the subclass 2 helper T-lymphocytes subset of activated T-lymphocytes produces interleukin (IL)-4, IL-5 and IL-13. IL-4 in conjunction with IL-13 signals the switch from IgM to IgE antibodies. The cross-linkage of two IgE molecules by allergen causes mast cells to degranulate, releasing histamine, leukotrienes, and other mediators that perpetuate the airway inflammation. IL-5 activates the recruitment and activation of eosinophils. The activated mast cells and eosinophils also generate their cytokines that help to perpetuate the inflammation.(Fireman 2003)
v During chronic inflammatory processes an excess of free radicals and DNA-reactive aldehydes from lipid peroxidation (LPO) are produced, which deregulate cellular homeostasis and can drive normal cells to malignancy.(Bartsch & Nair 2001)
Seri Kembangan, Malaysia
#5246 Feb 25, 2013
A study done by O'Byrne and Dalgleish in 2001 concluded that chronic inflammation may lead to the production of reactive oxygen species and metabolites such as malondialdehyde within affected cells that may in turn induce DNA damage and mutations and, as a result, be carcinogenic. They proposed that the conditions provided by a chronic inflammatory environment are so essential for the progression of the neoplastic process that therapeutic intervention aimed at inhibiting inflammation, reducing angiogenesis and stimulating cell mediated immune responses may have a major role in reducing the incidence of common cancers.
v Betulinic acid, a pentacyclic lupane-type triterpene, from Chaga (Inonotus obliquus): Chaga has been used in Eastern Europe, especially in Russia, as a folk medicine since the 16th century for treating cancer. Betulinic acid, a main compound found in Chaga powdered extract, is a selective inhibitor and inducer of apoptosis of many cancers (and HIV), including human melanoma. NF-κB inhibition has shown to be one of the many mechanisms through which betulinic acid suppresses melanoma and other cancers
o Cardiovascular Disease
v The process of inflammation is now believed to be the etiological event that precedes the development and the continual process of atherosclerosis. This process, beginning with an injury or change in the endothelial wall of the artery, causes an alteration in the intimal layer that increase leukocyte, LDL, and platelet adhesion to the endothelium. Possible causes of dysfunctional endothelium include: free radical damage from environmental exposure; hypertension and its proinflammatory effects (smooth muscle lipoxygenase activity and oxide radical formation; direct toxic effects of homocysteine; infections with Chlamydia pneumonia and herpes viruses; and advanced glycosylated end-products (the result of an oxidation reaction with glucose that results in a type of oxidant commonly found in the blood of diabetics).
v Most risk markers for cardiovascular disease have a pro-inflammatory component, which stimulates the release of a number of active molecules such as inflammatory mediators, reactive oxygen species (ROS), nitric oxide and peroxynitrite from endothelial, vascular smooth muscle and immune cells in response to injury.
v Risk factors for cardiovascular disease that have a pro-inflammatory component include LDL cholesterol, smoking, infection, homocysteine, ischemia, oxidant damage, interleukin-6, lipoprotein (a), high sensitivity C-reactive protein (hs-CRP), serum intracellular adhesion molecule-1 and apolipoprotein-B. One common link among these risk factors is the activity and metabolism of nitric oxide (NO).
Seri Kembangan, Malaysia
#5247 Feb 25, 2013
Oxidized or modified LDL is a recognized source or damage to the endothelial wall. The presence of oxidized LDL promotes the expression of growth factors and chemotactic proteins, causing an expanding inflammatory response and up-regulating monocyte replication to increase macrophage populations. The mediators of the inflammatory response: IL-1, TNF-α, IL-6, macrophage colony stimulating factor, etc, all increase the binding of LDL to the endothelium and smooth muscle to further up-regulate the inflammatory response.
v During the development of atherosclerosis, macrophages interact with vascular endothelial cells, medial smooth muscle cells and infiltrated inflammatory cells, particularly T cells and dendritic cells. Unquenched intracellular reactive oxygen species (ROS) induce monocytes to differentiate into macrophages. These cells then accumulate cholesterol esters in the cytoplasm, which leads to foam cell formation in lesion development.(Foam cells are fatty cells that together form the fatty streak the first identifiable characteristic lesion of advanced atherosclerosis).
v Further recruitment of monocytes and macrophages can occur by the release of cytokines from the endothelium and vascular smooth muscle as part of the inflammatory cycle. As a result, the atheromatous plaque core becomes rich in macrophages as the plaque ages. A vicious cycle ensues of endothelial cell activation that induces the expression of vascular cell adhesion molecule (VCAM-1) and monocyte chemo-attractant proteins (MCP-1), leading to increased monocyte/macrophage recruitment into the intima. This results in a soft plaque that increases the risk of unstable angina, thrombosis and acute myocardial infarction.
v Growing evidence indicates that chronic and acute overproduction of reactive oxygen species (ROS) under pathophysiologic conditions is integral in the development of cardiovascular diseases. ROS mediate various signaling pathways that underlie vascular inflammation in atherogenesis: from the initiation of fatty streak development through lesion progress to ultimate plague rupture.(Madamanchi 2004) When the plaque finally ruptures causing debris to barricade the artery it is disastrous.
v Data suggest that cytokine-induced endogenous iNOS (inducible nitric oxide synthase) expression and activity have key functions in increasing endothelial survival and maintaining function. Thus suppression of iNOS expression or limited substrate supply, as has been reported to occur in atherosclerosis patients, appears to significantly contribute to endothelial dysfunction and death during oxidative stress.(Hemmrich et al 2003)
v There is increasing evidence that systemic inflammation and insulin resistance constitute interrelated events that contribute to atherosclerosis. Circulating IL-6 levels, by inducing either hypertension in women or insulin resistance in men, constitutes a significant proatherogenic cytokine, as suggested by recent studies in which the serum IL-6 concentration was an independent predictor of cardiovascular mortality.(Fernandez-Real et al 2001)
v Inflammation (as quantified by CRP) appears to be associated with a significant prothrombotic status and endothelial dysfunction.(Apetrei 2004) CRP levels are markers of atheromatous plaque vulnerability and coronary artery disease activity.(Arroyo-Espliguero et al 2004)
v It can now be hypothesized that atherosclerosis may be an inflammatory disease that contributes to derangement of the vascular NO (nitric oxide) metabolic pathway and to increased oxidant stress. Recognizing that atherosclerosis is a multi-factorial inflammatory process lends to the assumption that anti-inflammatory nutrients might mitigate the disease.
Seri Kembangan, Malaysia
#5248 Feb 25, 2013
Inflammatory Bowel Disease
v Crohns disease and ulcerative colitis are chronic inflammatory bowel diseases of unknown origin. However the chronic immune stimulation of these conditions may be due to an imbalance of pro-inflammatory and inhibitory cytokines.(Lehmann 2003)
v It is well known that immune responses in the intestine remain in a state of controlled inflammation, suggesting that not only active suppression by regulatory T (TR) cells play an important role in the normal intestinal homeostasis, but also its dysregulation leads to the development of inflammatory bowel diseases.(Kanai & Watanabe 2004)
v The precise cause(s) of Crohns disease and ulcerative colitis are unknown. From animal models and human studies it is well established that gut bacteria flora are essential for inducing the bowel inflammation.(Chandran et al 2003)
v For more information on inflammatory bowel disease we invite you to read the section entitled "Inflammatory Bowel Disease, Crohn's Disease and Ulcerative Colitis" in our article on Gastro-Intestinal Disorders on our website.
o Insulin Resistance & Obesity
v Inflammation is a hyper-resistinemic state in humans, and cytokine induction of resistin may contribute to insulin resistance in endotoxemia, obesity and other inflammatory states. Adipocyte-derived resistin is a circulating protein implicated in insulin resistance in rodents, but the role of human resistin is uncertain because it is produced largely be macrophages.(Lehrke et al 2004)
v In a study done by Chen et al in 2004 they concluded that elevated levels of inflammatory markers are positively and independently associated with insulin resistance.
v Obesity and type 2 diabetes are associated with a state of abnormal inflammatory response. The state of chronic inflammation typical of obesity and type 2 diabetes occurs at metabolically relevant sites, such as the liver, muscle and most interestingly, adipose tissues. When TNF-alpha activity is blocked in obesity, either biochemically or genetically, the result is improved insulin sensitivity.(Hotamisligil 2003)
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