With a western diet, it is almost impossible to be deficient in calcium. SO this information is interesting but not applicable.BOTTOM LINE - CALCIUM INTAKE AND KIDNEY STONES:
1) Less calcium intake means more oxalate ions are free to enter the urine and bind with free calcium in solution forming stones.
2) Oxalate ions are more reactive than calcium ions and so the trick is to prevent free oxalate ions from entering the kidneys in excess amounts where they're most likely to bind with free calcium ions to form calcium oxalate stones.
3) Increasing dietary calcium intake would mean that most of the oxalate ions get compounded with the calcium and thus prevent a large number of free oxalate ions from entering the kidney.
4) Sodium intake should be minimized since sodium being more reactive than calcium (as sodium is higher up the activity series) can displace calcium ions from compounds there by causing free calcium ions to enter the kidneys where they can bond with free oxalate ions to form calcium oxalate crystals or kidney stones.
5) Intake of calcium supplements should be minimized since this form of calcium is SOMEHOW more likely to remain in the free state and so more likely to bond with free oxalate ions in the kidneys to form the calcium oxalate stones.
AFter a kidney stone episode, most urologists analyze your urine volume and composition. Composition will rule in or rule out metabolic issues. Volume will rule in if you need to drink more water. Furthermore, if you were lucky enoygh to capture the stone, sending it out to the lab is important to see which type it is, as not all are oxalates. A lot of what you read in the pop media (Volunterist for example) is actually CONTRAINDICATIVE if your stone is not an oxalate.
Obviously Volunteerist never had experience with a stone.